@themindthattellstroys: #inspiration #fyppppppppppppppppppppppp #Lifestyle #wallpaper

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Sunday 28 September 2025 22:10:59 GMT

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HE WAS JUST A BABY FOAL AND HE WAS DYING, SO WE TRIED OUR BEST TO FIX HIM Part IV of Flames Story “Listen to his little baby whinny! He’s got fight in him!!” “Flame is having a better day today,” Dr. Megan Marchitello told me excitedly. She was talking about Flame, the six to eight week old orphan colt who was dropped off at the slaughter holding facility for having birth defects and brain damage. “He greeted me with a happy little whinny. He loves his people. I got him talking to me the way he always does on a little video clip for you that I sent you, did you get it?” I told her I got it and we talked for a few minutes about how adorable he was before she got back to the medical talk. “I think today is the day we address his lax tendons.” She said. I knew this meant they were less worried about his other issues — failure to thrive, pneumonia and other contagious pathogens he was exposed to in the slaughter holding facility. Flame was born with a genetic defect called lax tendons. It can usually be corrected even by simple appropriate physical exercise but as Dr. Megan had pointed out, even Flame’s hind-end was totally atrophied. He either was orphaned at birth or his mother had not produced an appropriate amount of milk, or since he was an Amish buggy horse— it was possible she was taken away for hours and hours at a time for travel use for her owners. Flame would have been left on his own in the care of an eight year old boy. This time apart could cause malnutrition as foals need to feed frequently when they are young. This malnutrition had caused liver issues which had given him brain damage, to what degree and the lasting effect remained to be seen. Lax tendons in foals are a common condition, especially in premature or dysmature foals, where weak flexor tendons cause the fetlocks to sag. This can lead to symptoms like the toes pointing up and walking on the heels.

HE WAS JUST A BABY FOAL AND HE WAS DYING, SO WE TRIED OUR BEST TO FIX HIM Part IV of Flames Story “Listen to his little baby whinny! He’s got fight in him!!” “Flame is having a better day today,” Dr. Megan Marchitello told me excitedly. She was talking about Flame, the six to eight week old orphan colt who was dropped off at the slaughter holding facility for having birth defects and brain damage. “He greeted me with a happy little whinny. He loves his people. I got him talking to me the way he always does on a little video clip for you that I sent you, did you get it?” I told her I got it and we talked for a few minutes about how adorable he was before she got back to the medical talk. “I think today is the day we address his lax tendons.” She said. I knew this meant they were less worried about his other issues — failure to thrive, pneumonia and other contagious pathogens he was exposed to in the slaughter holding facility. Flame was born with a genetic defect called lax tendons. It can usually be corrected even by simple appropriate physical exercise but as Dr. Megan had pointed out, even Flame’s hind-end was totally atrophied. He either was orphaned at birth or his mother had not produced an appropriate amount of milk, or since he was an Amish buggy horse— it was possible she was taken away for hours and hours at a time for travel use for her owners. Flame would have been left on his own in the care of an eight year old boy. This time apart could cause malnutrition as foals need to feed frequently when they are young. This malnutrition had caused liver issues which had given him brain damage, to what degree and the lasting effect remained to be seen. Lax tendons in foals are a common condition, especially in premature or dysmature foals, where weak flexor tendons cause the fetlocks to sag. This can lead to symptoms like the toes pointing up and walking on the heels.

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